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A vlincRNA participates in senescence maintenance by relieving H2AZ-mediated repression at the INK4 locus
Sandra Lazorthes
Céline Vallot
Sébastien Briois
Marion Aguirrebengoa
Jean-Yves Thuret
Georges St. Laurent
Claire Rougeulle
1
Philipp Kapranov
Carl Mann
Didier Trouche
Estelle Nicolas
Sandra Lazorthes
Author
Céline Vallot
Author IdHAL : celine-vallot
Sébastien Briois
Author
Marion Aguirrebengoa
Author
Jean-Yves Thuret
Author
Georges St. Laurent
Author
Claire Rougeulle 1
Author
Philipp Kapranov
Author
Carl Mann
Author
Didier Trouche
Author IdHAL : didier-trouche ORCID : https://orcid.org/0000-0003-1398-6481
Estelle Nicolas
Author
1
EDC (UMR_7216) - Centre épigénétique et destin cellulaire (Université Paris Diderot Bât. Lamarck case postale 7042 75205 Paris CEDEX 13 - France)
- CNRS - Centre National de la Recherche Scientifique : UMR_7216 (France)
- UPC - Université Paris Cité : UMR_7216 (85 boulevard Saint-Germain 75006 Paris - France)
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Journal articles
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https://hal-cnrs.archives-ouvertes.fr/hal-03030452
Contributor : Mathieu Manquant Connect in order to contact the contributor
Submitted on : Monday, November 30, 2020 - 9:58:50 AM
Last modification on : Thursday, April 7, 2022 - 1:58:22 PM
Contributor : Mathieu Manquant Connect in order to contact the contributor
Submitted on : Monday, November 30, 2020 - 9:58:50 AM
Last modification on : Thursday, April 7, 2022 - 1:58:22 PM
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- HAL Id : hal-03030452, version 1
- DOI : 10.1038/ncomms6971
- PUBMEDCENTRAL : PMC4309439
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Citation
Sandra Lazorthes, Céline Vallot, Sébastien Briois, Marion Aguirrebengoa, Jean-Yves Thuret, et al.. A vlincRNA participates in senescence maintenance by relieving H2AZ-mediated repression at the INK4 locus. Nature Communications, Nature Publishing Group, 2015, 6 (1), ⟨10.1038/ncomms6971⟩. ⟨hal-03030452⟩
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