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Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells
Jhimmy Talbot
Raphael Peres
Larissa Pinto
Rene Oliveira
Kalil Lima
Paula Donate
Jaqueline Silva
Bernard Ryffel
1
Thiago Cunha
José Alves-Filho
Foo Liew
Paulo Louzada-Junior
Fernando Cunha
Fernando Cunha
Author
1
INEM - Immunologie et Neurogénétique Expérimentales et Moléculaires (Institut de Transgénose 3B, rue de la Férollerie 45071 Orléans Cedex 2 - France)
- UO - Université d'Orléans : UMR7355 (Château de la Source - Avenue du Parc Floral - BP 6749 - 45067 Orléans cedex 2 - France)
- CNRS - Centre National de la Recherche Scientifique : UMR7355 (France)
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Journal articles
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https://hal-cnrs.archives-ouvertes.fr/hal-02126580
Contributor : Valérie Quesniaux <>
Submitted on : Saturday, May 11, 2019 - 10:00:17 PM
Last modification on : Monday, August 12, 2019 - 3:16:08 PM
Contributor : Valérie Quesniaux <>
Submitted on : Saturday, May 11, 2019 - 10:00:17 PM
Last modification on : Monday, August 12, 2019 - 3:16:08 PM
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- HAL Id : hal-02126580, version 1
- DOI : 10.1186/s13075-018-1609-9
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Jhimmy Talbot, Raphael Peres, Larissa Pinto, Rene Oliveira, Kalil Lima, et al.. Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells. Arthritis Research and Therapy, BioMed Central, 2018, 20 (1), ⟨10.1186/s13075-018-1609-9⟩. ⟨hal-02126580⟩
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